Operation: HyperTrophy
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Operation: HyperTrophy - 6/10/2007 8:30:45 PM
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Creation
Posts: 436
Joined: 7/21/2006
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Workout: Hst Workout (8 week cycle but i skipped a week of 15's)
Supplements: 17-HD - 1 pill 30 min pre workout
Viraloid 2 pills 2x daily with food
Flaxseed 1 pill 3x daily
Fish Oil 2 pills 3x daily
CLA 1 pill 3x daily
B-complex 1pill 2x daily
AST multi 1 pill 2x daily (am/pm)
Bulk glutamine (5g post w/o and 5g before bed)
HMB 4 pills 3x daily (1000mg per 4 pills)
Vitamin C 1000mg post workout and 1000mg in the afternoon with a meal
Stats:
170 lbs 19. years old
6'1
Bicep: 13.5
Calf 15.5
Tricep: 12.2
Quad 21.25
Waist 32
Chest at nips 38
My goal for this cycle is nothing but hypertrophy or muscle size.
Nutrition:
Pre workout (30 min pre) - .1 g/lb whey , 17-hd
Pre ( .2 g/lbs of gatorade)
During (.2 g/lbs gatorade and a **** ton of water but not to much to feel blodded)
Postworkout ( .2 g/lbs whey, .4 g/lbs gatorade, 5g glutamine, 1000mg Vitamin C)
*(im working on getting some uni-liver and dextrose)
30-50min after shake i eat a solid meal.
like tuna, brown rice, and green beans.
My Eating guidelines i try for:
3-4 real meals a day and 2 supplement meals a day.
-only 2 fruits a day (before 3pm) always have one with first meal of the day.
-2-3 veggies a day
- only 10-20g carbs from sugar per meal
- 1 serving of almonds a day
-1.5 gallons of water a day
-no more than 15g fat per meal
and always a Casein shake w/ water before bed.
atleast 40g protein and 60g carbs per meal.
roughly 500-600 calories a meal.
Im currerntly on week 2 (1st week of my 10 reps) day 3 which was today.
Today's workout: 6/10
Warm up- 6 min on tredmill
Stretched super good
Squat (w/ warm up) 1x10 with 37.5 on each side (Felt good go up a bit more to hit max in 3 workouts)
Lunges( w/ warm up) 1x10 - 20 lb dumbells ( Form alot better this time, a lil to easy maybe?)
Leg curl 2x10 - 130 (good weight, cant wait to max out machine)
Incline Bench w/ dumbells(w/ warm up) 2x10 - 45lb dumbells (felt pretty good, awsome form)
Chins(w/ warm) 2x10 - (using assist machine) - weight at 55
Dips 1x10 - asisted machine at 25
rows 2x10 - 70lb ez bar
Side Lat raises 1x10 - 15lb dumbells (increment was to big of a jump only increase by 2.5)
right after side lat raise no rest- 1x10 calf raise w/ 60 lb dumbells
then immidetely into shrugs - 1x10 w/ 60 lb dumbells
Rear Lats raise - using the machine - 1x10 - 5lbs on each side
right after side lat raise no rest- 1x10 calf raise w/ 60 lb dumbells
then immidetely into shrugs - 1x10 w/ 60 lb dumbells
Shoulder Press w/ dumbells - 1x10 - 25lbs
BB curl w/ ez bar - 2x10 - had to do 1x10 with 60lbs then 1x10 with 50lbs
Decline oblique twist - 2x10 - 15lbs on chest.
Tricep Pushdowns- 2x10 - 60 lbs (next time only do 1x10 with 70 or mabye a 1x10 60 as well.)
Hyperextension(back extensions)- 2x10 - 35lb plate
finish up with 8-12 minutes of super intense Hiit.
on either the elliptical, bike, or tredmill.
This is my routine only thing that changes is sets and reps.
Ill have some comparision Pics when i finish as well.
Acutal Goals: Get up to 180.
Limit Drinking to once a week ( im a college student :(
No more THC before bed (im pretty sure it affects how much REM sleep you get which would maybe mean your muscles dont get the proper rest they need but this is just a theory from my own experiences.
Attain a V-taper that will actually be as wide as my hips. (my hips bones line up above my belly button aka they are huge and it sucks but my obliques have so much room to grow)
_____________________________
YOU GOTTA EAT BIG, TO BE BIG!
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RE: Operation: HyperTrophy - 6/11/2007 1:16:59 AM
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Creation
Posts: 436
Joined: 7/21/2006
Status: offline
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Found this interesting website about THC affecting REM sleep like i thought.
At low doses, marijuana may cause drowsiness, but at higher doses it seems to interfere with sleep. Specifically, it tends to decrease total REM time and also decrease eye movement during REM sleep.
http://www.madsci.org/posts/archives/dec97/875400410.Ns.r.html
_____________________________
YOU GOTTA EAT BIG, TO BE BIG!
(in reply to Creation)
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RE: Operation: HyperTrophy - 6/11/2007 1:20:30 AM
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Creation
Posts: 436
Joined: 7/21/2006
Status: offline
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Long-term adverse effects carry more serious consequences, but many people choose not to dwell on the long term. Using marijuana corresponds to the impairment of fetal growth and with a decreased length of gestation (Gurley et al. 1998). Developmental delays of the fetus also show up in pregnant women who use marijuana, but they have not been shown to be solely responsible. On the topic of lung damage that was addressed previously, marijuana contains more tar than cigarettes and, since most marijuana joints don’t contain filters, more of the particulates and carcinogens are inhaled. Thus, the potential for cancer and other lung diseases may be relatively high. Gynecomastia, the development of breast tissue in males, has also been cited as an adverse effect of marijuana. The condition is sensitive to changes in the ratio of estrogens to androgens which is thought to be altered by stimulation of the cytochrome P450 enzyme system by cannabis (Gurley et al. 1998). A frequently mentioned concern among smokers is that it might cause infertility, but this belief remains unconfirmed. Immunologic studies have, however, been used to show that cannabis acts as an immunosuppressant (Schlaadt et al. 1986). By inhibiting the body’s ability to respond to disease, it leaves the body more open to infections. The long-term effects of marijuana certainly seem more menacing than the short-term effects, but still don’t dampen the fact that marijuana is the most frequently used illicit drug in the United States.
http://sulcus.berkeley.edu/mcb/165_001/papers/manuscripts/_584.html
_____________________________
YOU GOTTA EAT BIG, TO BE BIG!
(in reply to Creation)
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RE: Operation: HyperTrophy - 6/11/2007 1:25:15 AM
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Creation
Posts: 436
Joined: 7/21/2006
Status: offline
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It was my assumption that the responses I would get would be attempts to rebutt the research. After all, this is a site for marijuana users. And the last 5 years I have worked with marijuana addicts, I have learned that any and all attempts will be made to rationalize and justify further use. I also know that the last one to perceive the connection between their use and the effects, even if it is clear to others. I can assure you that no drug can eliminate the need for REM sleep. That would be like saying a drug can eliminate the need for food and water. Here, to help get a clearer picture of marijuana's effects on the sleep cycle, here is part of my recent review on the research:
Introduction
Marijuana is the most commonly used illicit drug in the United States and is particularly common among young adults. In 2003, 57% of young adults (ages 19-28) who participated in the national Monitoring the Future (MTF) Study reported marijuana use at least once in their lifetime, and seventeen percent reported use within the last 30 days. Since so many young adults are potentially effected by marijuana use, research of the effects of marijuana use may be considered important. One area of marijuana research that has been explored since the 1970s is the effect of delta-9-tetrahydracannabinol (THC), the main psychoactive ingredient in marijuana, on sleep patterns (Florida Alcohol & Drug Abuse Association [FADAA], 2001). This paper summarizes some of the major research on this area from the 1970s to the present day.
Overview of the Sleep Cycle
Before exploring the effect of THC on sleep patterns, a brief review of the human sleep cycle may be helpful. Humans sleep in 5 different stages, differentiated primarily by brain wave patterns measured by electroencephalograph (EEG). Just prior to falling asleep, the typical person is in a relaxed state of consciousness characterized by alpha waves (a frequency of 8 to 12 waves per second). Just after falling asleep, phase 1 of the sleep cycle begins, during which brain activity is still fairly high but declining. Stage 2 is characterized by sleep spindles (12-to 14-Hz waves during a burst that lasts at least half a second) and K-complexes (sharp high-amplitude negative wave followed by a smaller, slower positive wave). The 3rd and 4th stages of sleep are known as slow-wave sleep (SWS). During SWS, heart rate, breathing rate, and brain activity slow down and the percentage of slow, large-amplitude waves increases. After stage 4, a person cycles back through stages 3 and 2. However, instead of returning to stage 1, the person enters a stage of sleep known as rapid eye movement (REM) sleep. REM sleep (also known as “paradoxical sleep”) is characterized by irregular, low-voltage fast brain waves. Despite this considerable degree of brain activity, the postural muscles of the body are more relaxed than at any other stage of sleep. Dreams are more vivid, intricate, and somewhat more frequent during this stage of sleep. Short-memory is consolidated into long-term memory. Since muscles are most relaxed during REM sleep, the body is best able to repair cells, and this function is vital to the immune system (Farthing, 1992; Kalat, 2001).
THC’s effect on SWS and REM sleep stages
Several studies have examined the effect of THC on SWS and REM sleep stages. In one of the earliest studies, rats injected with THC experienced induced bursts of polyspikes (on electrocortigram) just prior to stage 1 sleep. The polyspikes appeared again, overriding totally or partially the REM sleep stages (Masur & Khazan, 1970). Fujimori & Himwich (1973) found that THC caused a decrease in the number of REM sleep episodes in rabbits. By the third day of abstinence, the REM sleep cycle of the rabbits had returned to normal. Moreton & Davis (1973) measured the effect of THC on sleep cycles for both rats that were previously deprived of REM sleep and rats not REM-deprived, finding decreased SWS and REM sleep and increased wakefulness in rats injected with THC. Deniker, Ginestat, Etevenon, & Peron-Magnon (1975) found results verifying earlier research, with the added contribution of demonstrating that THC, when isolated from cannabis, has the same effect on sleep cycles as cannabis itself. The adverse impact of THC on SWS and REM sleep was noted in cats by Fairchild, Jenden, Mickey, & Yale (1979) and again in rats by Buonamici, Young, & Khazan (1982). Freemon (1982) conducted a study using two 23 and 25-year-old brothers, who slept in a laboratory for 27 nights and, following a 4 night break, 4 additional nights. THC administration and placebo administration was provided for both brothers alternately. The subjects experienced a decrease in SWS and REM sleep, and REM sleep had returned to normal about one week after abstinence from THC. However, it should be noted that the sample size for that study was very small. While examining the suspected anticonvulsant properties of 3 different cannabinoids on rats, Colasanti, Lindamood, & Craig (1982) found that both THC and delta-8-tetrahydracannibinol reduced REM sleep.
In the next decade of marijuana research, administration of THC was again found to decrease SWS and virtually eliminate REM stages in 11 cannabis naïve subjects aged 21 to 25 years (Tassinari, Ambrosetto, Peraita-Adrado, & Gestaut, 1999). However, Nicholson, Turner, Stone, & Robson (2004) found no effect of THC on nocturnal sleep on the first night of administration, unless administered in combination with cannabidiol, a non-psychoactive cannabinoid. Stage 3 sleep was decreased and wakefulness was increased in a group of 8 healthy 21-34-year-old subjects. However, THC administration by itself did reduce sleep latency on the 2nd day of administration.
Despite the research demonstrating THC’s tendency to decrease SWS and REM stage sleep, there is no shortage of individuals who claim that cannabis actually improves human sleep. Such individuals have cited a few sporadic studies that seem to them to confirm this hypothesis. For example, THC ingestion was actually found to decrease measures of sleep apnea (a breathing disorder that decreases restfulness) in rats by polysomonography analysis. However, this study did not examine the impact of THC on SWS or REM stages (Carley, Paviovic, Janelidze, & Radulovacki, 2002). Although it is possible that THC is effective in treating sleep apnea, the research is limited, and this possible relationship does not say anything about sleep quality. Sleep latency has also been examined for administration of THC in combination with sedative substances. For example, THC has been found to prolong pentobarbitone-induced sleep (Paton &Pertwee, 1972; Siemens et al., 1974) and ethanol- and hexobarbital-induced sleep (McCoy, Brown, & Forney, 1978). However, once again, the effect of THC on SWS and REM stages was not explored. Cannabidnol has been demonstrated to significantly decrease wakefulness and decrease SWS without significantly modifying REM sleep time (Siemens, 1974; Monti, 1977; Tassinari, Ambrosetto, Peraita-Adrado, & Gestaut, 1999). However, cannabidiol should not be confused with THC. Cannabidiol is not psychoactive and does not produce a “high,” nor does it bind to cannabinoid receptor sites (Mechoulam, Parker, & Gallily, 2002). Delta-8-tetrahydracannabinol has been found to induce sedation, enhance 12-hertz burst activity, and decrease the number of REM sleep episodes, while lengthening each REM episode, suggesting clinically useful sedative-hypnotic properties of this cannabinoid (Wallach & Gershon, 2002). However, delta-8-tetrahydracannabinol should not be confused with delta-9-tetrahydracannabinol (THC). One study, however, has resulted in the conclusion that THC significantly stabilizes respiration during all sleep stages, thus minimizing the adverse symptoms of sleep apnea. The suspected mechanism for this effect was cited as THC’s serotonin-inhibiting qualities (Carley, Paviovic, & Radulovacki, 2002). However, replications are needed to confirm these conclusions, and the possibility that THC stabilizes respiration does not say anything about sleep quality per se. Also, Rosenkrantz, Fleischman, & Grant (1981) have found that that THC actually caused dyspnea (breathing discomfort or significant breathlessness), among other health complications, in rhesus monkeys, adding some controversy to the equation. Page (1983), while conducting a correlational study on amotivational syndrome in marijuana users, did not find a difference in sleep EEG patterns between marijuana users and non-users in his Costa Rican sample. However, this does not appear to be a well-controlled experiment.
Other Effects of THC on Sleep
The combination of THC ingestion, stress, and REM deprivation has been shown to result in increased aggression in rats (Carlini, Lindsey, & Tufik, 1971; Carlini, 1977). In addition to adversely effecting SWS and REM cycles, withdrawal from THC use also appears to contribute to sleep problems. The 8 subjects in a study summarized in the preceding section experienced reduced sleep latency and changes in mood the day after THC ingestion (Nicholson, Turner, Stone, & Robson, 2004). Two subjects in the study conducted by Freemon (1982), summarized in the preceding section, experienced difficulty falling and staying asleep for the first 2 nights following a switch from THC ingestion to placebo ingestion. This 2 –day effect was also noted in the rats used by Colasanti, Lindamood, & Craig (1982) in the study noted in the previous section. Difficulty falling and staying asleep and restlessness was noted in 3 studies a few days after abstinence for both people who smoked marijuana and orally ingested THC (Budney, Moore, Vandrey, & Hughes, 2003; Haney et al., 1998a; Haney et al., 1998b). In addition to being commonly cited as a symptom of cannabis withdrawal, sleep disturbance and insomnia are often listed as an effect of long-term cannabis use (e.g. Beers, 2003; FADAA, 2001; Falkowski, 2000; Gold, 1989; Inaba & Cohen, 2003).
Discussion
The bulk of research in this area has demonstrated that THC has a negative impact on sleep quality both during use and during withdrawal, although much of it is outdated (1970s and 1980s) and more recent research is needed. Adolescence and early adulthood are regarded as periods of intense change. For some, these changes include transition to college, establishing a career, and independent living (Santrock, 1999). Young adults, especially students, often do not get adequate sleep. College students who report less sleep tend to not be as satisfied with life (Kelly, 2004). Stress, a lack of sleep, and substance abuse can lead to other problems for college students, including depression (Voelker, 2004). Given THC’s adverse impact on sleep quality, it can be concluded that avoidance of THC use would be an advantage for young adults.
Hope this is helpful.
http://www.marijuana.com/420/medicinal-marijuana/30780-sweet-dreams-marijuana-dreams-rem.html
_____________________________
YOU GOTTA EAT BIG, TO BE BIG!
(in reply to Creation)
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RE: Operation: HyperTrophy - 6/12/2007 3:23:45 PM
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Creation
Posts: 436
Joined: 7/21/2006
Status: offline
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awsome eating day and rest day yesterday. Today ill be doing the setup but everything up a bit more then 1 more day and i get to finally see how strong ive gotten from the 15's and 10's alone.
_____________________________
YOU GOTTA EAT BIG, TO BE BIG!
(in reply to Creation)
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